Prozac, the New Wonder Drug for the Treatment of Cancer and Leukemia
Prozac is a serotonin uptake inhibitor which is used in the treatment of depression. When serotonin is increased in a neuronal synapse, neuronal activity is increased resulting in a decrease in depression symptoms.
For example, Prozac (fluoxetine) is well known to protect neurons against different forms of stress induced damage.
http://www.ncbi.nlm.nih.gov/entrez/queryd.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstract&list_uids=15456934&itool=pubmed_docsum
http://www.ncbi.nlm.nih.gov/entrez/queryd.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstract&list_uids=11522596&itool=pubmed_docsum
The fundamental question is HOW?
Apparently, Prozac (fluoxetine) is a major anti-inflammatory drug that inhibits the synthesis of dangerous pro-inflammatory immune hormones by blocking the activation of NF-kappaB, the genetic factor that controls the synthesis of most inflammatory hormones, survival factors that prevent programmed cell death in cancer and leukemia cells, while enhancing the synthesis of membrane pumps that extrude chemotherapy drugs from cancer cells as quickly as they enter.
Effective NF-kappaB inhibitors are few and far between. Inhibiting the activity of this genetic factor is one of the primary goals of all cancer therapies. Prozac in normal doses may be the answer to many of our problems.
Remember, in a culture dish Prozac enhances the efficacy of 5 major cancer chemo drugs, including Taxol, by 10 to 100 times. It does this by blocking the activity of the membrane pumps. This results in a greater accumulation of chemo drugs in the cancer cell. We now know that the inhibition of membrane pump synthesis is due to Prozac's ability to block NF-kappaB signaling.
http://grouppekurosawa.com/blog/2009/01/prosac-anti-depressant-drug
Who knew?
Stay tuned...
Grouppe Kurosawa, Medicine in the Public Interest
http://www.grouppekurosawa.com
Everything considered, Prozac is a great drug for the treatment of depression. However, over time it has become obvious that Prozac has other biochemical properties that transcend its ability to regulate serotonin uptake into neurons.
For example, Prozac (fluoxetine) is well known to protect neurons against different forms of stress induced damage.
http://www.ncbi.nlm.nih.gov/entrez/queryd.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstract&list_uids=15456934&itool=pubmed_docsum
http://www.ncbi.nlm.nih.gov/entrez/queryd.fcgi?db=pubmed&cmd=Retrieve&dopt=Abstract&list_uids=11522596&itool=pubmed_docsum
The fundamental question is HOW?
Apparently, Prozac (fluoxetine) is a major anti-inflammatory drug that inhibits the synthesis of dangerous pro-inflammatory immune hormones by blocking the activation of NF-kappaB, the genetic factor that controls the synthesis of most inflammatory hormones, survival factors that prevent programmed cell death in cancer and leukemia cells, while enhancing the synthesis of membrane pumps that extrude chemotherapy drugs from cancer cells as quickly as they enter.
Effective NF-kappaB inhibitors are few and far between. Inhibiting the activity of this genetic factor is one of the primary goals of all cancer therapies. Prozac in normal doses may be the answer to many of our problems.
Remember, in a culture dish Prozac enhances the efficacy of 5 major cancer chemo drugs, including Taxol, by 10 to 100 times. It does this by blocking the activity of the membrane pumps. This results in a greater accumulation of chemo drugs in the cancer cell. We now know that the inhibition of membrane pump synthesis is due to Prozac's ability to block NF-kappaB signaling.
http://grouppekurosawa.com/blog/2009/01/prosac-anti-depressant-drug
Who knew?
Stay tuned...
Grouppe Kurosawa, Medicine in the Public Interest
http://www.grouppekurosawa.com
posted by Dr. Steve at 10:31 AM
1 Comments:
You have the similar thing on this link but less agressive
http://www.lowdosenaltrexone.org/index.htm
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